Hypothesis: type I toxin - antitoxin genes enter the persistence field-a feedback mechanism explaining membrane homoeostasis
Research output: Contribution to journal › Journal article › Research › peer-review
Original language | English |
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Article number | 20160189 |
Journal | Royal Society of London. Philosophical Transactions B. Biological Sciences |
Volume | 371 |
Issue number | 1707 |
Number of pages | 9 |
ISSN | 0962-8436 |
DOIs | |
Publication status | Published - 2016 |
- bacterial persistence, toxin, antitoxin, HokB, RNase E, membrane homoeostasis
Research areas
ID: 170195227