Hypothesis: type I toxin - antitoxin genes enter the persistence field-a feedback mechanism explaining membrane homoeostasis
Research output: Contribution to journal › Journal article › Research › peer-review
| Original language | English |
|---|---|
| Article number | 20160189 |
| Journal | Royal Society of London. Philosophical Transactions B. Biological Sciences |
| Volume | 371 |
| Issue number | 1707 |
| Number of pages | 9 |
| ISSN | 0962-8436 |
| DOIs | |
| Publication status | Published - 2016 |
- bacterial persistence, toxin, antitoxin, HokB, RNase E, membrane homoeostasis
Research areas
ID: 170195227